plasmids paav ctnt icre (Addgene inc)
93
Structured Review
Addgene inc
plasmids paav ctnt icre
Plasmids Paav Ctnt Icre, supplied by Addgene inc, used in various techniques. Bioz Stars score: 93/100, based on 9 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/plasmids+paav+ctnt+icre/pAAV%2EcTNT%2EiCre+(Plasmid+%2369916)/pmc12926986-65-1-5
Average 93 stars, based on 9 article reviews
Plasmids Paav Ctnt Icre, supplied by Addgene inc, used in various techniques. Bioz Stars score: 93/100, based on 9 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/plasmids+paav+ctnt+icre/pAAV%2EcTNT%2EiCre+(Plasmid+%2369916)/pmc12926986-65-1-5
Average 93 stars, based on 9 article reviews
plasmids paav ctnt icre - by Bioz Stars,
2026-10
93/100 stars
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Related Articles
CRISPR:Article Title: Lztr1 deficiency contributes to the pathogenesis of dilated cardiomyopathy via RAP1/PI3K/AKT-mediated myocardial Injury. Article Snippet: LZTR1 is a member of the BTB-Kelch protein family and participates in various cellular processes.. Existing studies have reported its association with myocardial injury, which aligns with our clinical case observations, though its precise pathogenic mechanisms remain incompletely understood.. Clinical investigations revealed that patient harboring pathogenic LZTR1 mutations displayed markedly diminished LZTR1 protein expression, which was clinically associated with progressive dilated cardiomyopathy (DCM) and decompensated heart failure (HF). Knockdown:Article Title: Lztr1 deficiency contributes to the pathogenesis of dilated cardiomyopathy via RAP1/PI3K/AKT-mediated myocardial Injury. Article Snippet: LZTR1 is a member of the BTB-Kelch protein family and participates in various cellular processes.. Existing studies have reported its association with myocardial injury, which aligns with our clinical case observations, though its precise pathogenic mechanisms remain incompletely understood.. Clinical investigations revealed that patient harboring pathogenic LZTR1 mutations displayed markedly diminished LZTR1 protein expression, which was clinically associated with progressive dilated cardiomyopathy (DCM) and decompensated heart failure (HF). |